Spirochetes and MS

This spirochete was isolated from the grey matter of a an MS patient in Germany in 1922 by Dr. Gabriel Steiner who invented Steiner-Silver-Stain, a stain we still use today. His work on MS has been all but forgotten.
Thank you goes to: Thomas Grier

Magnetic resonance imaging signatures of vascular pathology in multiple sclerosis.

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Utriainen D, Trifan G, Sethi S, Elias S, Hewett J, Feng W, Haacke EM.

Source

Magnetic Resonance Innovations, Inc., Detroit, MI, USA.

Abstract

Venous vascular contributing factors to multiple sclerosis (MS) have been known for some time. Only recently has the scope of their potential role become more apparent with the theory of chronic cerebrospinal venous insufficiency (CCSVI). As research expands to further explore the role of vascular pathology in the MS population, it is expedient to review the evidence from an imaging perspective. In this paper, we review the current state-of-the-art methods using magnetic resonance imaging (MRI) as applied to imaging MS patients and CCSVI. This includes evaluating imaging signatures of vascular structure and flow as well as brain iron content. Upon review of the literature, we find that extracranial venous anomalies including stenosis, venous malformations, and collateralization of flow in the major veins of the neck have been observed to be prevalent in the MS population. Abnormal flow has been reported in MS patients both in major vessels using phase-contrast flow quantification and in the brain using perfusion-weighted imaging. We discuss the role of quantitative flow imaging and its potential in assessing possible biomarkers for abnormal flow. Finally, it has been suggested that the presence of high iron content may indirectly indicate progression of existing vascular pathology. To that end, we review the use of susceptibility-weighted imaging in monitoring iron in the thalamus, basal ganglia, and MS lesions.

PMID:

22971468

[PubMed - in process]

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Multiple sclerosis is Lyme disease: Anatomy of a cover-up

Multiple sclerosis is Lyme disease: Anatomy of a cover-up

Perhaps the biggest ongoing medical scandal of the past hundred years is the fact that it has been known since 1911 that Multiple Sclerosis is caused by a bacterium, and that the medical establishment covered this up, in order to make money selling symptom relievers to MS patients. Since 1911, overwhelmingly much medical research has been conducted where living Borrelia bacteria were found in the brains of people who were diagnosed with MS.

Time and time again. By at least a dozen medical researchers. In at least ten countries. Since 1911 – the past one hundred years. Several older but also recent autopsy findings linked to in this article found that all deceased MS patients’ brains harbored living Lyme spirochetes. Even when tests, notorious for their large percentage of false negatives were used on living MS patients, staggeringly many tested positive for active Lyme borreliosis.

Then why isn’t this common knowledge? Surely, those thousands of MS experts and MS researchers can’t be all wrong?

Let’s examine the reality on the ground.

1. Multiple Sclerosis Societies.

Every Western country has at least one MS Society. Each of those tax-exempt societies typically receives tens of millions of dollars in funding from various sources, year after year. The people running those societies usually award themselves CEO-level salaries and run them as one would run a highly commercial corporation. Advertising is used to solicit funds but if you don’t read ads then you’ll bump into them, one day, begging you for money on the street. For all those billions that have been pumped over the decades in those hundreds of MS societies worldwide, not a single one has ever done anything really useful for MS patients. The worst that could possibly happen for the bosses of those setups is that the cause of MS would become known. A known cause would either mean the development of either a cure or at least better symptom relievers, and that would rapidly result in the obsoleteness of their money making machine – the chicken that lays the golden eggs if you will. Such MS societies are working in concert with MS “researchers” employed by Big Pharma.

2. Big Pharma.

Multinational pharmaceutical corporations are the only ones doing MS research nowadays, mainly using donations to MS societies. Those multinationals decide which researchers get the cash. Researchers wanting to test the postulation of bacterial etiology of MS are shunned as if they were crackpots. Big Pharma makes billions a year on MS symptom relievers and they trickle millions down to their footsoldiers, the “MS experts”. A cure would be a severe financial blow. Even more so, because there is strong evidence that many other neurological illnesses are caused by germs as well. Because due to the phenomenon of immune privilege there is an inadequate immune response in the brain and spinal cord, making these organs the ideal place for certain slow-dividing spirochetal bacteria to entrench, multiply and cause lesions. The entire concept of antibiotic-resistant, hard-to-test-for chronic CNS infections leading to a dearth of neurological syndromes has to be suppressed and what can’t be suppressed will be craftily discredited. Better to give every expression of a neurological infection its own name such as “MS”, “Alzheimers”, “Parkinsons,” “ALS” and “Fibromyalgia”. And fund armies of ignorant “experts” to obfuscate the issue, whilst boycotting, firing, censoring, smearing and suing those few real experts that refuse to stay in line. Big Pharma is in business to make money, and money is made when people are ill, not when they’re healthy. Anyone standing in their way is relegated to the sidelines. Patents are being bought and shelved so that cures will never see the light of day.

3. Patient advocacy groups.

MS patient groups are, without exception, populated with clueless individuals for the simple reason that those who did their homework and read the relevant research have been ostracized by the group. They always were and they always will, because that’s how group dynamics works. As soon as you insist on voicing an opinion outside of the mainstream, no matter how well argued – you’ll be an outcast, a pariah. They don’t want rogue activists, “lone nutters”, giving them a bad name. Also the advocacy groups are raking in the dough and are run by folks whose main concern is that membership dues are paid in time. No MS, no advocacy group. Of course if there ever will emerge a lobby group insisting on more microbiological research pertaining Multiple sclerosis, they’ll be branded “lunatic fringe” and their efforts will be in vain.

4. MS “experts”.

Those “experts” get away with calling themselves thus, because Big Pharma gives them their seal of approval in the form of research grants and medical media exposure. However they are only experts in doing exactly what Big Pharma wants them to do: Obscuring the cause of Multiple Sclerosis! In return, the “experts” get regular cash injections for their “promising research” and other goodies such as all-in holidays to exotic destinations. There never will be a cure for MS until the scandal breaks and new antibiotics are developed that work better than the few currently available antibiotics that cross the blood-brain barrier. As it stands, it has been more than twenty years ago since any new antibiotic was developed. As soon as it was found that Minocycline helped with MS, its manufacturer, Lederle, tripled its price.

After long consideration I came to the conclusion that at least a crucial part of this debâcle was due to a real conspiracy – mainly a conspiracy of silence of those few MS researchers bright enough to realize that the cause for MS has been known for at least a hundred years. As is always the case with medical cover-ups, it continues to exist due to a mix of ignorance, indifference, cowardice and corruption.  The saying goes: “Do not attribute to malice that what can be adequately blamed on ignorance”. All the “experts” really are interested in is being “experts”, not curing Multiple sclerosis.  However it still is a conspiracy. It is completely normal for conspiracies to succeed because the lion share of the people who could point it out don’t care, are too lazy to get educated or feel too intimidated to stick out their necks. Microbiologist Tom Grier calls them cowards. The fact that most conspiracies are silently facilitated by an army of “useful idiots” with a stake in it being kept under the rug does not make it any less a conspiracy.

Evidence for a conspiracy of silence

Now I’ve given my opinion. You may find it harsh – I call it mild.

You don’t have to believe me, when I say there is a conspiracy. Believe Alzheimer and Parkinson’s disease expert Dr. Alan B. MacDonald M.D., Staff Pathologist at the St. Catherine of Siena Medical Center. He wrote:

(published online 10 July 2006 in Volume 67, Issue 4, page 819-832 in Medical Hypotheses)

“Conventional thinking about spirochetal cyst forms is divided between two polar spheres of influence; one a majority community that completely denies the existence of spirochetal cyst forms, and a second group of academically persecuted individuals who accepts the precepts of such antebellum scientists as Schaudinn, Hoffman, Dutton, Levaditi, Balfour, Fantham, Noguchi, McDonough, Hindle, Steiner, Ingraham, Coutts, Hampp, Warthin, Ovcinnikov, and Delamater. Microscopic images of cystic spirochetes are difficult to ignore, but as has been the case in this century, academic “endowments” have nearly expunged all cystic spirochetal image data from the current textbook versions of what is the truth about the spirochetaceae. If the image database from the last century is obliterated; many opportunities to diagnose will be lost. Variously sized cystic spirochetal profiles within diseased nerve cells explain the following structures: Lewy body of Parkinson’s disease, Pick body, ALS spherical body, Alzheimer plaque. Borrelia infection is therefore a unifying concept to explain diverse neurodegenerative diseases, based not entirely on a corkscrew shaped profile in diseased tissue, but based on small, medium and large caliber rounded cystic profiles derived from pathogenic spirochetes which are hiding in plain sight.”

Note how he claims that the majority of researchers deny the existence of spirochetal cystic forms. Denial is defined as knowing that something exists, but deliberately refusing to acknowledge it for ulterior motives.  By putting “endowments” between question marks, he implies that Big Pharma bribes universities and publishers into censoring the very existence of spirochetal cysts from medical textbooks.

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Chlaymdial Pneumonia cause for MS?

Dr Wheldon believes he put his wife in remission from her MS but treating her for an infection called, Chlamydial Pneumonia. Could this be the key to MS?

Click Here for Transcription of the interviews with Dr David Wheldon.

Bartonella mimicks MS

Abstract 

From Clinical Neurology and Neurosurgery
Volume 112, Issue 7 , Pages 625-628, September 2010
Rare infections mimicking MS
Vesna V. Brinar,
Mario Habek

The diagnosis of multiple sclerosis (MS), despite well defined clinical criteria is not always simple. On many occasions it is difficult to differentiate MS from various non-MS idiopathic demyelinating disorders, specific and infectious inflammatory diseases or non-inflammatory demyelinating diseases. Clinicians should be aware of various clinical and MRI “red flags” that may point to the other diagnosis and demand further diagnostic evaluation. It is generally accepted that atypical clinical symptoms or atypical neuroimaging signs determine necessity for broad differential diagnostic work up. Of the infectious diseases that are most commonly mistaken for MS the clinician should take into account Whipple's disease, Lyme disease, Syphilis, HIV/AIDS, Brucellosis, HHV-6 infection, Hepatitis C, Mycoplasma and Creutzfeld-Jacob disease, among others. Cat scratch disease caused by Bartonella hensellae, Mediterranean spotted fever caused by Riketssia connore and Leptospirosis caused by different Leptospira serovars rarely cause focal neurological deficit and demyelinating MRI changes similar to MS. When atypical clinical and neuroimaging presentations are present, serology on rare infectious diseases that may mimic MS may be warranted. This review will focus on the infectious diseases mimicking MS with presentation of rare illustrative cases.

Keywords: Multiple sclerosis, Red flags, MRI, Infections

Chlamydial Pneumonia, MS Cause?

Multiple sclerosis: a chronic infective cerebrospinal venulitis?

 Abstract

The aetiology proposed for the development of chronic cerebrospinal venous insufficiency (CCSVI) associated with multiple sclerosis (MS) has been the presence of congenital truncular venous malformations. However, this hypothesis is not consistent with the epidemiology or geographical incidence of MS and is not consistent with many of the ultrasonographic or radiographical findings of the venous disturbances found in MS patients. However, the probability of a venous aetiology of MS remains strong based on evidence accumulated from the time the disorder was first described.

The method used in this review was to search PubMed for all past medical publications related to vascular, venous, haematological, epidemiological, biochemical, and genetic investigations and treatments of MS.

Epidemiological and geographical findings of prevalence of MS indicate the involvement of an infective agent. This review of the venous pathology associated with MS describes a hypothesis that the pathogenesis of the venous disease could be initiated by a respiratory infective agent such as Chlamydophila pneumonia, which causes a specific chronic persistent venulitis affecting the cerebrospinal venous system. Secondary spread of the agent would initially be via the lymphatic system to specifically involve the azygos, internal jugular and vertebral veins. The hypothesis proposes mechanisms by which an infective venous vasculitis could result in the specific neural damage, metabolic, immunological and vascular effects observed in MS. The hypothesis described is consistent with many of the known facts of MS pathogenesis and therefore provides a framework for further research into a venous aetiology for the disease.

If MS does result from a chronic infective venulitis rather than a syndrome involving congenital truncular venous malformations, then additional therapies to the currently used angioplasties will be required to optimize results. 

http://phleb.rsmjournals.com/content/27/5/207.short

Charts Comparing MS, Lyme, Fibro and Chronic Fatigue

Click Pictures to Enlarge